An infectious disease that was eradicated in 1977. The medical establishment continues to credit the smallpox vaccine for eliminating smallpox, but the vaccine was actually a failure. Mortality rates from smallpox increased after compulsory vaccinations from 2.04 per 10,000 individuals in 1850 to 10.24 in 1871. Leicester, England had one of the highest vaccination rates in the vaccinated world and their smallpox breakout was higher than ever. They decided to stop the smallpox vaccine, and once they did they had the lowest rate of smallpox infection and deaths in the world.
Every vaccine has a story behind it, Dr. Suzanne Humphries, author of Dissolving Illusions: Disease, Vaccines, and the Forgotten History, says. The smallpox vaccine, for example, was actually developed long before the medical establishment knew anything about the human immune system. The revelations on smallpox alone are fascinating enough to purchase this book, and is far more detailed than the summary in this article.
The vaccine was actually developed based on a rumor circulating among dairy maids. The rumor was that when a dairy maid had been infected with cowpox—which is a common infection on the udder of the cow—she would no longer be susceptible to smallpox. The rumor was a persistent one, as rumors can be, despite the fact that there were plenty of dairy maids who developed smallpox after having cowpox. But this rumor is what led Edward Jenner to develop the first smallpox vaccine.
“Basically, it was made by scraping pus off the belly of a cow,” Dr. Humphries says. “Sometimes there was some goat genetic disease in there. There was horsepox mixed in there.
There was sometimes human pox mixed in and some glycerin. They would shake it up; they would take kind of a prong, and puncture the skin several times…
What I didn’t realize was that there were many people who developed serious smallpox disease and died after they were vaccinated. The severity of disease was often worse in the vaccinated than the unvaccinated.
There are statistics that show that the death rate was higher in the vaccinated than the unvaccinated.”
When the smallpox vaccine was developed, there was also no way to accurately diagnose the type of pox disease a person had. It may have been chickenpox, monkeypox, or smallpox, but back then, any kind of pox disease was considered smallpox—even though the vaccine didn’t actually have the human smallpox virus in it. Animal pox virus was always used. According to Dr. Humphries, it was the most contaminated vaccine that’s ever been on the market.
“If you look at a town like Leicester in England, that town was noticing that they had one of the highest vaccination rates in the vaccinated world and their smallpox breakout was higher than ever,” Dr. Humphries says. “The people in the town had a rally. The mayor and some of the health officials were there. They all agreed that they were going to stop vaccinating… The result was quite different from the predictions.
The predictions were that there was going to be a bonfire of disease set upon the planet and that these people in Leicester were risking the health of the world by not making vaccination mandatory. But once they stopped smallpox vaccines they had the lowest rate of smallpox infection and deaths.
What we show in our book – and we show the graphs of the disease rates and the death rates – was that both of them went down precipitously after the vaccinations were stopped. That story right there tells you that vaccines were not what made the disease go away; what made the disease go away was isolation and sanitation.”
Antibody Is the Wrong Way to Ascertain Immunity
One of the major arguments against vaccine-induced immunity is that it primarily stimulates the humoral immune system and not the cellular immune system. Antibodies are produced by the humoral immune system and then routinely measured to determine “immunity.” The problem with this approach is that you can have high antibody levels and still get the disease. It’s very difficult and expensive to measure the cellular immune response, and immunologists admit that they are still in the dark about a lot of the finer points of the overall immune response.
When you use antibody titers or blood levels to check for immunity, all you’re doing is getting a picture of what happened (you had an immune response); it doesn’t tell you whether you’re going to be immune in the future, because antibodies are only one aspect of the immune response, and in some cases are not even necessary to easily combat the sickness and become immune.
For example, those with agammaglobulinemia—a disease where you cannot make antibodies — can get infected with measles, recover uneventfully, and still respond to subsequent challenges of the virus in a normal healthy fashion and not get sick. These individuals will have lifelong immunity to measles, the same as someone without agammaglobulinemia.
Traditionally, the way immunity is determined is to do a test that measures antibodies, which is the humoral immune system. But there’s no good way to assess the cellular immune system. It’s a really imprecise science at best. As Dr. Humphries notes:
“It’s not only imprecise; sometimes it’s downright inaccurate. You can have very high antibody levels, like numerous case reports of people who have hugely high antibody levels for tetanus, or normal antibodies, and have gotten some of the worst cases of tetanus. I have papers that show that people without antibody for polio have actually been able to respond to the virus as if they were already immune. The antibody really is a real wrong roadmap to look at to tell what’s really going on. Sometimes there’s correlation, but it’s certainly not a given.”